Myoclonus After Cardiac Arrest

Myoclonus After Cardiac Arrest - Web circumstances of cardiac arrest: Prognostication after cardiac arrest often depends primarily on neurological function, and characterizing the extent of neurological injury hinges on neurophysiological testing and clinical neurological examination. Differentiation between these two entities is therefore critically important, because they have different prognostic implications. The presence of early posthypoxic myoclonus (phm) following cardiac arrest had been invariably associated with poor. Acute — starts within 48 hours after a cardiac arrest.

Prognostication after cardiac arrest often depends primarily on neurological function, and characterizing the extent of neurological injury hinges on neurophysiological testing and clinical neurological examination. Web circumstances of cardiac arrest: Differentiation between these two entities is therefore critically important, because they have different prognostic implications. The presence of early posthypoxic myoclonus (phm) following cardiac arrest had been invariably associated with poor. Acute — starts within 48 hours after a cardiac arrest.

Differentiation between these two entities is therefore critically important, because they have different prognostic implications. Acute — starts within 48 hours after a cardiac arrest. Web circumstances of cardiac arrest: Prognostication after cardiac arrest often depends primarily on neurological function, and characterizing the extent of neurological injury hinges on neurophysiological testing and clinical neurological examination. The presence of early posthypoxic myoclonus (phm) following cardiac arrest had been invariably associated with poor.

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Acute — Starts Within 48 Hours After A Cardiac Arrest.

Web circumstances of cardiac arrest: Differentiation between these two entities is therefore critically important, because they have different prognostic implications. The presence of early posthypoxic myoclonus (phm) following cardiac arrest had been invariably associated with poor. Prognostication after cardiac arrest often depends primarily on neurological function, and characterizing the extent of neurological injury hinges on neurophysiological testing and clinical neurological examination.

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